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Poster Presentations
Day 2, June 23（Mon.）
Room P (Maesato East, Foyer, Ocean Wing)
- 2P-AM-20
Multi-omics analyses reveal the potential mechanism of nematode resistance in tomato resistant and susceptible cultivars
(1DBBS, NCKU, 2CSD, TARI, 3CGRBD, TARI)
oYing-An Chen1, Le Kang2, Yuan-Kai Tu3, Ying-Lan Chen1
Root-knot nematodes (RKN) pose a significant threat to agriculture, causing yield reductions of up to 40% in infected tomatoes. Many commercial tomato varieties carrying the Mi-1 locus can combat RKN infection. However, breeding nematode-resistant tomatoes requires a long period, and genetically modified approaches also face regulatory restrictions in many countries. Thus, we explored the mechanism of the Mi-1 locus and investigated alternative methods to enhance plant resistance to nematodes. In this study, we determine the Mi/Mi (resistant) or mi/mi (susceptible) genotype of tomato plants using specific genotyping markers. Using both Mi/Mi and mi/mi cultivars, we applied quantitative proteomic analyses to understand potential proteins involved in nematode resistance. Additionally, we recently found a plant peptide hormone that can enhance resistance against RKN in a commercial heat-tolerant tomato cultivar (CL5915) lacking the Mi-1 gene. To assess whether tomatoes lacking the Mi-1 locus can resist nematodes when treated with the peptide, we performed a proteomic analysis on the mi/mi cultivar infected with RKN with or without CAPE1 pre-treatment. The peptide-induced proteome changes will be discussed in relation to potential mechanisms and methods of acquired resistance to nematodes. This investigation aims to provide deeper insights into resistance mechanisms and suggest strategies for mitigating nematode impacts on tomato plants.